Example sentences of "see in patients " in BNC.

  Next page
No Sentence
1 Professor Wolf and his colleagues point out that all these diseases may show symptoms of nerve degeneration which are similar to those found in ABL and can also be treated by supplements of vitamin E. For example , a striking reduction in axonal degeneration has been seen in patients treated with vitamin E , for pancreatic insufficiency associated with cystic fibrosis .
2 Furthermore , the overall gain in time to progression and length of survival was seen in patients born with and without tumour related symptoms .
3 Hence the neurologists of the late nineteenth and early twentieth centuries , having recognised that various different patterns of language disorder existed , went on to attempt to define these as syndromes — for example , to define several different categories of aphasia , each category being defined as a list of the symptoms that are seen in patients belonging to that category .
4 Schwartz ( 1985 ) has discussed ways in which the model of normal sentence production proposed by Garrett ( 1980 , 1981 , 1982 ) , which we described in Chapter 8 , might be used to interpret defects of sentence production seen in patients with any form of expressive aphasia .
5 This general approach can also be used to interpret the patterns of impairment to language processing seen in patients with acquired disorders of language produced by neurological damage .
6 Rises in whole body turnover rates have also been seen in patients with inflammatory bowel disease .
7 The patients positive to cow 's milk challenge also mounted a specific antibody secreting cells response against gliadin in the IgM class ( Fig 3 ) , which was not seen in patients negative to challenge ( Fig 4 ) .
8 The increased proliferation and the significant upwards trend of the proliferating cells towards the apex of the crypt , seen in patients with adenomatous polyps , is a product of the increased S-phase duration of the pre-malignant cells and may be specifically related to cancer .
9 The degree of acid reflux in association with CLO is greater than that seen in patients with reflux oesophagitis as a whole .
10 The prolonged oesophageal acid exposure seen in patients with hiatus hernia is because of reduced acid clearance but it is only seen in patients with lower oesophageal sphincter deficiency .
11 The prolonged oesophageal acid exposure seen in patients with hiatus hernia is because of reduced acid clearance but it is only seen in patients with lower oesophageal sphincter deficiency .
12 The increase in synthesis of LTB 4 seen in patients taking NSAIDs remained significant when the subgroups who were Helicobacter pylori positive or negative were analysed separately .
13 By contrast there were no significant differences in synthesis of LTC 4 in NSAID users between those with type C gastritis ( 2.3 ( 1.5–3.0 ) pg/mg ) compared with those with normal mucosa ( 1.8 ( 1.2–2.8 ) pg/mg ) although enhanced synthesis in those with type B gastritis ( 3.2 ( 1.5–4.0 ) pg/ mg ) reflected the higher concentrations seen in patients colonised with Helicobacter pylori ( p<0.02 ) .
14 The confounding use of second line drugs does not explain the increased synthesis of LTB 4 seen in patients taking NSAIDs overall .
15 Synthesis of LTB 4 by gastric mucosal epithelial cells has been reported and it is possible that these are an important source of the increased concentrations we have seen in patients taking NSAIDs .
16 Furthermore , fundic argyrophil endocrine tumours developed in five of 17 — that is , 29.5% of patients with associated MEN 1 while none was seen in patients with sporadic type disease .
17 Both types of stomach are seen in patients with duodenal ulcer diseas and this is relevant to the surgical treatment of the condition since patients with acid antra may be inadequately treated by highly selective vagotomy .
18 The increase in PGE 2 associated with Helicobacter pylori seems likely to be a consequence of the associated inflammatory cell infiltrate as it was seen in patients with type B ( chronic active ) but not with type C ( chemical ) gastritis , which is characterised by a paucity of inflammatory cells .
19 The enhanced synthesis of PGE 2 seen in patients taking NSAIDs and infected with Helicobacter pylori might mediate the increased dyspapsia that characterises these patients .
20 Figure I shows the most common pattern of prolonged contraction seen in patients with incontinence — that is , a strong initial sphincter contraction followed by rapid relaxation .
21 Our PSS patients rported reflux symptoms , however , only during a small fraction of the acid exposure time , as previously seen in patients with chest pain or gastro-oesophageal reflux .
22 Duodenal H pylori was only seen in patients with gastric metaplasia ( 10 ( 50% ) receiving NSAIDs , and 34 ( 89% ) not receiving NSAID ) .
23 Chronic active duodenitis was diagnosed when intramucosal neutrophils were seen in patients scoring four or more .
24 Similar numbers of duodenal ulcers ( 25–30% ) were also seen in patients with active duodenitis or gastric metaplasia , or both , regardless of NSAID intake .
25 Microcytosis is also seen in patients with anaemia of chronic disease , the thalassaemia syndromes and , rarely , sideroblastic anaemia .
26 Sporadic cases are seen in patients in their 20s or early 30s .
27 A surprising observation is that the thrombosed grafts were confined to the patients who received a low dose of aprotinin ; graft occlusions were not seen in patients allocated to receive a high dose or placebo .
28 While research dealing with the optimum length of hospital stay is sparse , significantly fewer relapses are seen in patients who complete an inpatient programme and are discharged at a normal weight than in those who leave prematurely .
  Next page